Brief reportHistiocytoid cardiomyopathy: A cause of sudden death in apparently healthy infants
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Cited by (45)
2019 HRS expert consensus statement on evaluation, risk stratification, and management of arrhythmogenic cardiomyopathy
2019, Heart RhythmCitation Excerpt :Glycogen, lipids, and pigment may be observed in these cells, as well as a lymphocytic infiltrate. Immunostaining shows perimembranous immunoreactivity for muscle-specific actin, but not for the histiocytic markers, S-100 protein and CD69.387,391,398,402,403 These cells may be abnormal Purkinje cells; however, a primitive myocardial precursor cannot be excluded.
Histiocytoid cardiomyopathy in an eleven-month-old infant: A case report and literature review
2017, Human Pathology: Case ReportsCitation Excerpt :Cell proliferation markers are usually negative. The cytoplasm of histiocytoid cells contains a large amount of mitochondria and lipid droplets of variable size but lacks a T-tubule system [20,25–28]. The histiocytoid cells also show poorly developed intercellular junctions.
Conduction Defects/Cardiomyopathies
2014, Advances in PediatricsCitation Excerpt :Glycogen, lipid, and pigment may be seen in these cells as well as a lymphocytic infiltrate. Immunostaining shows perimembranous immunoreactivity for muscle-specific actin but not for the histiocytic markers, S100 protein and CD69 (KP) [65–69]. These cells may be abnormal Purkinje cells, but a primitive myocardial precursor cannot be excluded.
Demonstration of MyoD1 expression in oncocytic cardiomyopathy: Report of two cases and review of the literature
2004, Pathology Research and PracticeHistiocytoid cardiomyopathy: A cause of sudden death in infancy
1997, Pathology Research and Practice
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On leave of absence from the Department of Pathology, Washington University, St. Louis, Missouri.