Brief report
Histiocytoid cardiomyopathy: A cause of sudden death in apparently healthy infants

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Cited by (45)

  • 2019 HRS expert consensus statement on evaluation, risk stratification, and management of arrhythmogenic cardiomyopathy

    2019, Heart Rhythm
    Citation Excerpt :

    Glycogen, lipids, and pigment may be observed in these cells, as well as a lymphocytic infiltrate. Immunostaining shows perimembranous immunoreactivity for muscle-specific actin, but not for the histiocytic markers, S-100 protein and CD69.387,391,398,402,403 These cells may be abnormal Purkinje cells; however, a primitive myocardial precursor cannot be excluded.

  • Histiocytoid cardiomyopathy in an eleven-month-old infant: A case report and literature review

    2017, Human Pathology: Case Reports
    Citation Excerpt :

    Cell proliferation markers are usually negative. The cytoplasm of histiocytoid cells contains a large amount of mitochondria and lipid droplets of variable size but lacks a T-tubule system [20,25–28]. The histiocytoid cells also show poorly developed intercellular junctions.

  • Conduction Defects/Cardiomyopathies

    2014, Advances in Pediatrics
    Citation Excerpt :

    Glycogen, lipid, and pigment may be seen in these cells as well as a lymphocytic infiltrate. Immunostaining shows perimembranous immunoreactivity for muscle-specific actin but not for the histiocytic markers, S100 protein and CD69 (KP) [65–69]. These cells may be abnormal Purkinje cells, but a primitive myocardial precursor cannot be excluded.

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On leave of absence from the Department of Pathology, Washington University, St. Louis, Missouri.

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