Effects of intravenous adenosine on verapamil-sensitive “idiopathic” ventricular tachycardia
References (26)
- et al.
Atrial induction of ventricular tachychardia: reentry versus triggered automaticity
Am J Cardiol
(1979) - et al.
Ventricular tachycardia induced by atrial stimulation in patients without symptomatic cardiac disease
Am J Cardiol
(1983) - et al.
Idiopathic recurrent sustained ventricular tachycardia responsive to verapamil: an ECG-electrophysiologic entity
Am Heart J
(1984) - et al.
Exercise provocable right ventricular outflow tract tachycardia
Am Heart J
(1982) - et al.
Use of intravenous verapamil for ventricular tachycardia
Am J Cardiol
(1984) - et al.
Adenosine in the diagnosis of broad-complex tachycardia
Lancet
(1988) - et al.
Relative efficacy of various physical manoeuvres in the termination of junctional tachycardia
Lancet
(1988) - et al.
Comparison of adenosine and verapamil for termination of paroxysmal junctional tachycardia
Am J Cardiol
(1989) - et al.
The value of the electrocardiogram in the differential diagnosis of a tachycardia with a widened QRS complex
Am J Med
(1978) - et al.
Observations on mechanisms of ventricular tachycardia in man
Circulation
(1976)
Continuous local electrical activity: a mechanism of recurrent ventricular tachycardia
Circulation
(1978)
Response of recurrent sustained ventricular tachycardia to verapamil
Br Heart J
(1981)
Idiopathic paroxysmal ventricular tachycardia with a QRS pattern of right bundle branch block and left axis deviation: a unique clinical entity with specific properties
Am J Cardiol
(1983)
Cited by (67)
2017 AHA/ACC/HRS Guideline for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death: A Report of the American College of Cardiology/American Heart Association Task Force on Clinical Practice Guidelines and the Heart Rhythm Society
2018, Journal of the American College of CardiologyDrug Therapy in Adult Congenital Heart Disease
2017, Cardiac Electrophysiology ClinicsCitation Excerpt :It can also slow down the AV node during arrhythmias that are not AV nodal dependent (such as atrial fibrillation, atrial flutter, and atrial tachycardia) revealing the underlying P or fibrillatory waves. It usually has no effect on ventricular tachycardia except outflow tract VT which is triggered-activity mediated.41 It has a peak effect in 10 seconds and its effect lasts for approximately 10 more seconds.
ESC 2015 guidelines for the treatment of patients with ventricular arrhythmias and prevention of sudden cardiac death
2016, Revista Espanola de CardiologiaFascicular tachycardia
2014, Cardiac Electrophysiology ClinicsA complex rhythm treated simply: Fascicular ventricular tachycardia
2014, American Journal of Medicine
Copyright © 1994 Published by Elsevier Inc.