ArticleSacral transcutaneous oxygen tension levels in the spinal cord injured: Risk factors for pressure ulcers?
References (34)
Acute pressure area care: Sir James Paget's legacy
Lancet
(1992)- et al.
Anaerobic metabolism and wound healing: hypothesis for initiation and cessation of collagen synthesis in wounds
Am J Surg
(1978) - et al.
Transcutaneous oxygen tension in selection of amputation level
Am J Surg
(1984) - et al.
The prevention and rehabilitation of ischemic ulcers
- et al.
Prevalence and incidence of pressure sores in acute spinal cord injuries
Paraplegia
(1981) - et al.
Prevention of pressure sores by identifying patients at risk
Br Med J
(1982) - et al.
Risk factors for early-occurring pressure ulcers following spinal cord injury
Am J Phys Med Rehab
(1988) Etiology and pathology of ischemic ulcers
Arch Phys Med Rehabil
(1959)Decubitus ulcers: role of pressure and friction in causation
Arch Phys Med Rehabil
(1974)- et al.
The pressure sore: pathophysiology and principles of management
Ann Intern Med
(1981)
Decubitus ulcers
J Am Board Fam Pract
Etiologic factors in presure sores: experimental model
Arch Phys Med Rehabil
Spinal cord injuries: comprehensive management and research
Adrenergic receptors in insensitive skin of spinal cord injured patients
Arch Phys Med Rehabil
New principles of resuscitation for brain and spinal injury
N Carolina Med J
Catecholaminergic content of peripheral plasma in human subjects with complete transverse lesions of the spinal cord
J Physiol
Plasma catecholamines during paroxysmal neurogenic hypertension in quadriplegic man
Circ Res
Cited by (32)
Changes in sympathetic neurovascular function following spinal cord injury
2018, Autonomic Neuroscience: Basic and ClinicalCitation Excerpt :Therefore another possible explanation for impairment of sensory nerve mediated-vasodilation is spinal reflex activation of sympathetic-vasoconstrictor axons in SCI subjects (Kuesgen et al., 2002). The transcutaneous oxygen tension levels measured in sacral skin (covering the base of the spine where pressure sores commonly develop) have been reported to be lower in subjects with SCI, indicating poor nutritive blood flow in the superficial layers of the skin (Mawson et al., 1993). Furthermore, as SCI lowers BP, blood flow in skin can be occluded with lower externally applied pressures than in able-bodied subjects (Schubert and Fagrell, 1991).
Reproducibility of Transcutaneous Oxygen Pressure Measurements in Persons With Spinal Cord Injury
2009, Archives of Physical Medicine and RehabilitationA repeat audit of spinal board usage in the emergency department
2008, InjuryCitation Excerpt :One study identified that the incidence of pressure sores over the bony prominences can be related back to prolonged time spent on the spinal board whilst waiting for investigations.6 Shocked patients have a reduced perfusion pressure and those with spinal injuries with potential neurological deficit in auto-regulation of blood pressure and loss of pain sense are at greater risk of sores.7,8 Ironically, it is these patients that are likely to be placed on boards in the first instance and are likely to stay on them longer due to complicated management plans.
Cardiovascular consequences of loss of supraspinal control of the sympathetic nervous system after spinal cord injury
2000, Archives of Physical Medicine and RehabilitationCitation Excerpt :Similar arteriovenous shunting has been reported in individuals with quadriplegia.139 Transcutaneous oxygen tension is reportedly significantly lower in individuals with SCI than in controls, especially in the supine position.140 As mentioned previously, individuals with high cord injuries suffer reduced resting blood pressure.