Trends in Pharmacological Sciences
Volume 17, Issue 4, April 1996, Pages 141-144
ReviewExcitatory actions of adenosine on ventricular automaticity
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Cited by (9)
The increase in rat ventricular automaticity induced by salbutamol is mediated through β<inf>1</inf>- but not β<inf>2</inf>-adrenoceptors: Role of phosphodiesterases
2011, Life SciencesCitation Excerpt :Abnormal automaticity is an important mechanism underlying cardiac arrhythmias, and cAMP increases the spontaneous firing rate of pacemaker cells (DiFrancesco, 2010). The right ventricle of the rat contains pacemaker cells in the His-Purkinje fibers, which develop spontaneous activity resembling an idioventricular rhythm (Mangoni and Nargeot, 2008) and is a useful experimental model for assessing the pro-arrhythmic effect of cAMP producing agents (Hernandez et al., 1994; Hernandez and Ribeiro, 1996). The aims of the present study were to evaluate the effects of β1- and β2-AR activation on ventricular automaticity in this tissue and to examine whether these effects were modified by theophylline or PDE3–4 inhibition.
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