Brief reportDelayed presentation of an epidural hematoma
References (27)
- et al.
Acute bilateral epidural hematoma
Surg Neurol
(1985) - et al.
Traumatic bilateral epidural hematomas presenting at different times in a patient with large ventricles
Surg Neurol
(1985) - et al.
Sequential angiographic studies demonstrating delayed development of an acute epidural hematoma
J Neurosurg
(1978) - et al.
Serial computed tomographic scanning and the prognosis of severe head injury
Neurosurgery
(1979) - et al.
Neurological course and correlated computerized tomography findings after severe closed head injury
J Neurosurg
(1980) - et al.
The value of serial computerized tomography in the management of severe head injury
Surg Neurol
(1979) - et al.
The value of computerized tomography in the management of 1000 consecutive head injuries
Surg Neurol
(1977) - et al.
Delayed intracranial hematoma in patients with severe head injury
Radiology
(1979) - et al.
Delayed traumatic intracerebral haemorrhage
J Neurol Neurosurg Psychiatry
(1972) Delayed traumatic intracerebral hematomas after surgical decompression
Neurosurgery
(1979)
Delayed traumatic intracerebral hematomas
J Neurosurg
Delayed epidural hemorrhage in hypotensive multiple trauma patients
Neurosurgery
Delayed epidural hematoma demonstrated by computed tomography: Case report
Neurosurgery
Cited by (10)
Extrapleural hematoma that was not detectable on initial CT in a patient with pneumonia
2021, American Journal of Emergency MedicineCitation Excerpt :Although our patient had developed pain of sudden onset suggestive of hemorrhage, EH was not detected on the first CT examination but was identified when CT was repeated 33 h later to investigate the ongoing pain. Similar to delayed epidural hematoma [28], delayed EH takes time to become obvious on CT. Although the tissue between the parietal pleura and innermost intercostal muscles consists of loose connective tissue and fat [1], the tissue between the innermost and internal intercostal muscles is denser, so even a small amount of bleeding that is not visible on CT could cause severe pain (Fig. 3).
Posttraumatic Triple Acute Epidural Hematomas: First Report of Bilateral Synchronous Epidural Hematoma and a Third Delayed
2020, World NeurosurgeryCitation Excerpt :The most supported hypothesis about the mechanism responsible for the formation of a DEDH concerns, above all, the loss of a tamponade effect given by the reduction of intracranial pressure (ICP) with medical or surgical treatment and develops preferentially on the contralateral side and with a concomitant skull fracture.6 The loss of this tamponade effect, caused by reduction of intracranial pressure due to edema treatment with hyperventilation and/or hyperosmotic diuretics, decompressive craniectomy, or removal of intracranial bleedings, has been proposed in the literature as a determining factor in the development of a DEDH.7-9 The reduction of ICP after those procedures should favor bleeding, which determines the DEDH.
Delayed, but acutely progressive epidural hematoma after mild head injury [8]
2001, American Journal of Emergency MedicineCitation Excerpt :Delayed epidural hematoma (DEH) has been extensively described in the literature,1-9 and has been defined as “an epidural hematoma that is not present in the first neuroradiographic examination made after trauma, but that appears in sequential neuroradiologic explorations during patient evolution.”1 In most reported cases of DEH, the initial “normal” computed tomography (CT) scans are taken within a few hours after injury, and evolution of the “delayed” hematoma is completed within 1 to 4 days after injury.1-8 The authors report a rare case of a DEH in a young man who presented with rapidly deteriorating level of consciousness caused by an epidural hematoma 6 days after a mild head injury, although his brain CT scan 2 days after the injury was normal.