Clinical practiceFailure of new biochemical markers to exclude acute myocardial infarction at admission
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Optimal design of nanowire field-effect troponin sensors
2017, Computers in Biology and MedicineThe prognostic and risk-stratified value of heart-type fatty acid-binding protein in septic patients in the emergency department
2014, Journal of Critical CareCitation Excerpt :It appears in the blood as early as 1.5 hours after onset of symptoms of myocardial injury, peaks around 6 hours, and returns to baseline values in 24 hours [9]. These features of H-FABP make it superior to traditional biomarkers for the assessment of recurrent or persistent myocardial damage [10–12]. Subsequent studies have shown that H-FABP is produced and secreted by other tissues, except the myocardium, including the skeletal muscle, kidney, gut, and brain, whereas ischemic injury and H-FABP correspond to the severity of injury [13].
Cardiac biomarkers in acute myocardial infarction
2013, International Journal of CardiologyCitation Excerpt :cTn was proposed as a specific marker of myocardial necrosis but the high sensitivity of cTn compared with CK and CKMB had not been envisaged. Early studies showed that cTn was raised in AMI (as diagnosed by WHO criteria) [7,20–27] with high sensitivities and specificities [7,12,17,22,24,25,28–38] and had the advantage over CKMB in differentiating cardiac from skeletal muscle injury [7,39]. Studies in the 1990s also showed that significant numbers of patients classed as unstable angina (as opposed to AMI) by conventional WHO criteria, had elevated cTn levels [25,26,36,38–44].
Biochemical markers in acute coronary syndrome
2011, Clinica Chimica ActaDevelopment of a plasma panel test for detection of human myocardial proteins by capillary immunoassay
2007, Biosensors and Bioelectronics