Liver damage in heatstroke
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Cited by (156)
Liver in Systemic Disease
2023, MacSween's Pathology of the Liver, Eighth EditionSheng Mai San ameliorated heat stress-induced liver injury via regulating energy metabolism and AMPK/Drp1-dependent autophagy process
2022, PhytomedicineCitation Excerpt :The liver is one of the most sensitive organs in hot environment. HS leads to severe damage to the liver, which is validated by liver dysfunction, oxidative stress, inflammatory response and distortion in hepatic parenchyma (Davis et al., 2017; Kew et al., 1970). At present, due to HS is becoming an increasing morbidity and mortality burden, no safe and effective therapies for HS are available in clinics.
Dietary supplementation of camel whey protein attenuates heat stress-induced liver injury by inhibiting NLRP3 inflammasome activation through the HMGB1/RAGE signalling pathway
2021, Journal of Functional FoodsCitation Excerpt :Liver injury has been identified as a fatal complication in clinical cases and animal models of HS (Geng et al., 2015; Kew, Bersohn, Seftel, & Kent, 1970; Hassanein, Razack, Gavaler, & Van Thiel, 1992; Wang et al., 2018). Even if treatments to lower body temperature are utilized, they can fail to prevent the occurrence of liver damage in HS cases, which in turn leads to death (Kew et al., 1970; Weigand, Riediger, Stremmel, Flechtenmacher, & Encke, 2007). Recent studies have shown that in HS-treated rat models, liver injury is not observed during HS, but gradually becomes severe during recovery from HS (Geng et al., 2015; Leon, 2007).
Liver transplantation in patients with liver failure related to exertional heatstroke
2019, Journal of HepatologyHeat stress induced alternative splicing in catfish as determined by transcriptome analysis
2019, Comparative Biochemistry and Physiology - Part D: Genomics and Proteomics
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From the Department of Medicine, University of the Witwatersrand and Johannesburg General Hospital, Johannesburg, the South African Institute for Medical Research, Johannesburg, and the Department of Pathology, Northwestern University and Wesley Memorial Hospital, Chicago, Illinois. This study was supported by a grant from the South African Chambers of Mines.